Search results for "Caveolin 1"

showing 10 items of 17 documents

VEGF-R2/Caveolin-1 Pathway of Undifferentiated ARPE-19 Retina Cells: A Potential Target as Anti-VEGF-A Therapy in Wet AMD by Resvega, an Omega-3/Poly…

2021

Age-related macular degeneration (AMD) is one of the main causes of deterioration in vision in adults aged 55 and older. In spite of therapies, the progression of the disease is often observed without reverse vision quality. In the present study, we explored whether, in undifferentiated ARPE-19 retinal cells, a disruption of the VEGF receptors (VEGF-R)/caveolin-1 (Cav-1)/protein kinases pathway could be a target for counteracting VEGF secretion. We highlight that Resvega®, a combination of omega-3 fatty acids with an antioxidant, resveratrol, inhibits VEGF-A secretion in vitro by disrupting the dissociation of the VEGF-R2/Cav-1 complex into rafts and subsequently preventing MAPK activation.…

0301 basic medicineMAP Kinase Signaling SystemAngiogenesisQH301-705.5Caveolin 1Drug Evaluation PreclinicalresveratrolResveratrolAMDRetinaArticleCatalysisCell LineVEGF-receptorInorganic ChemistryMacular Degeneration03 medical and health scienceschemistry.chemical_compoundangiogenesis0302 clinical medicinemedicineHumansSecretionPhysical and Theoretical ChemistryBiology (General)Molecular BiologyQD1-999SpectroscopyRetinaomega-3 fatty acidsKinaseOrganic Chemistryocular diseasesRetinalGeneral MedicineVascular Endothelial Growth Factor Receptor-2VEGFIn vitroComputer Science ApplicationsTranscription Factor AP-1Chemistry030104 developmental biologymedicine.anatomical_structurechemistry030220 oncology & carcinogenesisCaveolin 1Cancer researchInternational Journal of Molecular Sciences
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Paracrine effects of human adipose-derived mesenchymal stem cells in inflammatory stress-induced senescence features of osteoarthritic chondrocytes

2016

Aging and exposure to stress would determine the chondrocyte phenotype in osteoarthritis (OA). In particular, chronic inflammation may contribute to stress-induced senescence of chondrocytes and cartilage degeneration during OA progression. Recent studies have shown that adipose-derived mesenchymal stem cells exert paracrine effects protecting against degenerative changes in chondrocytes. We have investigated whether the conditioned medium (CM) from adipose-derived mesenchymal stem cells may regulate senescence features induced by inflammatory stress in OA chondrocytes. Our results indicate that CM down-regulated senescence markers induced by interleukin-1β including senescence-associated β…

0301 basic medicineSenescenceAgingPathologymedicine.medical_specialtyadipose-derived mesenchymal stem cells conditioned mediumsenescenceCaveolin 1chondrocytesAdipose tissueInflammationmedicine.disease_cause03 medical and health sciencesParacrine signalling0302 clinical medicineOsteoarthritisParacrine CommunicationmedicineHumansCellular SenescenceInflammation030203 arthritis & rheumatologybiologySirtuin 1KinaseMesenchymal stem cellMesenchymal Stem CellsCell Biologybeta-GalactosidaseCell biologyOxidative Stress030104 developmental biologyAdipose Tissuebiology.proteinmedicine.symptomOxidative stressResearch PaperAging
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Pravastatin reverses the membrane cholesterol reorganization induced by myocardial infarction within lipid rafts in CD14(+)/CD16(-) circulating monoc…

2012

International audience; Large numbers of monocytes are recruited in the infarcted myocardium. Their cell membranes contain cholesterol-rich microdomains called lipids rafts, which participate in numerous signaling cascades. In addition to its cholesterol-lowering effect, pravastatin has several pleiotropic effects and is widely used as secondary prevention treatment after myocardial infarction (MI). The aim of this study was to investigate the effects of pravastatin on the organization of cholesterol within monocyte membrane rafts from patients who had suffered myocardial infarction. Monocytes from healthy donors and acute MI patients were cultured with or without 4μM pravastatin. Lipid raf…

AdultMalemedicine.medical_specialtyStatinmedicine.drug_classCD14[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionCaveolin 1Lipopolysaccharide Receptors030204 cardiovascular system & hematologyCD16GPI-Linked ProteinsMonocytes03 medical and health scienceschemistry.chemical_compound0302 clinical medicineCaveolaeInternal medicinemedicineHumansMolecular BiologyLipid raftCells Cultured030304 developmental biologyPravastatin0303 health sciencesCholesterolMonocyteAnticholesteremic AgentsReceptors IgGstatinCell BiologyMiddle Aged3. Good healthlipid raftEndocrinologymedicine.anatomical_structureCholesterolmyocardial infarctionchemistryGene Expression RegulationImmunologycaveolaemonocyteFemalelipids (amino acids peptides and proteins)[SDV.AEN]Life Sciences [q-bio]/Food and NutritionPravastatinmedicine.drug
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Expression of caveolin-1 in tooth germ, ameloblastoma and ameloblastic carcinoma

2020

Background The caveolin-1 protein (structural component of membrane caveolae) plays important roles in several biological functions, such as endocytosis, cell adhesion, and cell signaling. However, this protein has been associated with mechanisms of tumorigenesis in several neoplasms. The expression patterns and roles of caveolin-1 in the oral epithelium and in embryonic and odontogenic tumor tissues are still unclear. Material and Methods The expression of caveolin-1 was evaluated in samples of the normal gingival epithelium (n=7), human tooth germ (TG) (n=12), ameloblastoma (AM) (n=83), and ameloblastic carcinoma (AC) (n=9) by immunohistochemistry. Additionally, AM samples were analyzed b…

AdultPathologymedicine.medical_specialtyReal Time Polymerase Chain Reactionrecurrencegovernment.form_of_governmentCaveolin 1Odontogenic TumorsBiologymedicine.disease_causeAmeloblastic CarcinomaMalignant transformationAmeloblastomaWestern blotCaveolin-1medicineHumansCaveolina 1resectionAmeloblastomaGeneral DentistrycurettageUNESCO:CIENCIAS MÉDICASdesmoplastic ameloblastomaOral Medicine and Pathologymedicine.diagnostic_testResearchmixed radiopaque ? radiolucentCarcinomaOdontogenic tumorTooth Germmedicine.diseaseImmunohistochemistryAmeloblastic carcinomaOtorhinolaryngologyCaveolin 1governmentImmunohistochemistrySurgeryCarcinogenesisenucleationMedicina Oral, Patología Oral y Cirugía Bucal
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Chronic peroxisome proliferator-activated receptorβ/δ agonist GW0742 prevents hypertension, vascular inflammatory and oxidative status, and endotheli…

2015

Endothelial dysfunction plays a key role in obesity-induced risk of cardiovascular disease. The aim of the present study was to analyze the effect of chronic peroxisome proliferator-activated receptor (PPAR)β/δ agonist GW0742 treatment on endothelial function in obese mice fed a high-fat diet (HFD).Five-week-old male mice were allocated to one of the following groups: control, control-treated (GW0742, 3 mg/kg per day, by oral gavage), HFD, HFD + GW0742, HFD + GSK0660 (1 mg/kg/day, intraperitoneal) or HFD-GW0742-GSK0660 and followed for 11 or 13 weeks. GW0742 administration to mice fed HFD prevented the gain of body weight, heart and kidney hypertrophy, and fat accumulation. The increase in …

Blood GlucoseMaleAgonistmedicine.medical_specialtyNitric Oxide Synthase Type IIIEndotheliumPhysiologymedicine.drug_classCaveolin 1Peroxisome proliferator-activated receptorThiophenesDiet High-FatGW0742MiceInsulin resistanceInternal medicineInternal MedicinemedicineAnimalsObesityPPAR deltaSulfonesEndothelial dysfunctionReceptorPPAR-betaAortachemistry.chemical_classificationInterleukin-6Tumor Necrosis Factor-alphabusiness.industryGlucose Tolerance TestPeroxisomemedicine.diseaseToll-Like Receptor 4VasodilationThiazolesEndocrinologymedicine.anatomical_structureAdipose TissuechemistryHypertensionAdiponectinEndothelium VascularInsulin ResistanceReactive Oxygen SpeciesCardiology and Cardiovascular MedicinebusinessJournal of Hypertension
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Plasma membrane and lysosomal localization of CB1 cannabinoid receptor are dependent on lipid rafts and regulated by anandamide in human breast cance…

2005

AbstractIn this report we show, by confocal analysis of indirect immunofluorescence, that the type-1 cannabinoid receptor (CB1R), which belongs to the family of G-protein-coupled receptors, is expressed on the plasma membrane in human breast cancer MDA-MB-231 cells. However, a substantial proportion of the receptor is present in lysosomes. We found that CB1R is associated with cholesterol- and sphyngolipid-enriched membrane domains (rafts). Cholesterol depletion by methyl-β-cyclodextrin (MCD) treatment strongly reduces the flotation of the protein on the raft-fractions (DRM) of sucrose density gradients suggesting that CB1 raft-association is cholesterol dependent. Interestingly binding of …

CB1 receptorCannabinoid receptorMESH: Membrane MicrodomainsMESH: Receptor Cannabinoid CB1Biochemistrychemistry.chemical_compoundRaftsMESH: Cholesterol0302 clinical medicineReceptor Cannabinoid CB1Structural BiologyReceptorLipid raft0303 health sciencesChemistrybeta-CyclodextrinsAnandamideEndocannabinoid system3. Good healthCell biologyCholesterollipids (amino acids peptides and proteins)AgonistMESH: beta-CyclodextrinsMESH: Cell Line TumorPolyunsaturated Alkamidesmedicine.drug_classBiophysicsBreast NeoplasmsArachidonic Acids03 medical and health sciencesMembrane MicrodomainsCell Line TumorGeneticsmedicineMESH: Arachidonic AcidsHumansMolecular Biology030304 developmental biologyG protein-coupled receptorMESH: HumansMESH: Polyunsaturated AlkamidesCell Membrane[SDV.BBM.BM]Life Sciences [q-bio]/Biochemistry Molecular Biology/Molecular biologyAnandamideCell BiologyCaveolin 1LysosomesIntracellular traffickingMESH: Breast Neoplasms030217 neurology & neurosurgeryMESH: Cell MembraneMESH: LysosomesEndocannabinoids
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Tumorigenic conversion of endothelial cells.

2003

Tumors of endothelial origin develop rarely. Until now, only two angiosarcoma (AS)-derived endothelial cell lines have been be isolated, ISO-HAS and AS-M. Both AS-derived endothelial cell lines presented the typical endothelial characteristics, such as the expression of CD31 and von Willebrand factor, but differed from normal endothelial cells in a nuclear expression of p53, in a delayed angiogenic reaction, and a reduced expression of caveolin. In addition, differences in the expression of cytokines and cell adhesion molecules responsive to proinflammatory stimuli were observed. While AS-M showed an expression pattern similar to that of human umbilical vein endothelial cells (HUVEC), ISO-H…

CD31AdultLipopolysaccharidesTelomerasePathologymedicine.medical_specialtyClinical BiochemistryCaveolin 1Vascular Cell Adhesion Molecule-1BiologyCaveolinsPathology and Forensic Medicinevon Willebrand FactormedicineCell AdhesionHumansMolecular BiologyTelomeraseCells CulturedCell NucleusCell adhesion moleculeReverse Transcriptase Polymerase Chain ReactionGranulocyte-Macrophage Colony-Stimulating FactorTelomereIntercellular Adhesion Molecule-1Cell biologyVascular endothelial growth factor BEndothelial stem cellDNA-Binding ProteinsPlatelet Endothelial Cell Adhesion Molecule-1Vascular endothelial growth factor ACell Transformation NeoplasticVascular endothelial growth factor CCell cultureEndothelium VascularTumor Suppressor Protein p53Experimental and molecular pathology
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Caveolin and GLT-1 gene expression is reciprocally regulated in primary astrocytes: Association of GLT-1 with non-caveolar lipid rafts

2004

Caveolae represent membrane microdomains acting as integrators of cellular signaling and functional processes. Caveolins are involved in the biogenesis of caveolae and regulate the activity of caveolae-associated proteins. Although caveolin proteins are found in the CNS, the regulation of caveolins in neural cells is poorly described. In the present study, we investigated different modes and mechanisms of caveolin gene regulation in primary rat astrocytes. We demonstrated that activation of cAMP-dependent signaling pathways led to a marked reduction in protein levels of caveolin-1/-2 in cortical astrocytes. Application of transforming growth factor-alpha (TGF-alpha) also resulted in a decre…

Central Nervous SystemCaveolin 2Caveolin 1Down-RegulationGlutamic AcidBiologyCaveolinsHistone DeacetylasesChromatin remodelingRats Sprague-DawleyPhosphatidylinositol 3-KinasesCellular and Molecular NeuroscienceAstrocyte differentiationMembrane MicrodomainsCaveolaeCaveolinCyclic AMPAnimalsRNA MessengerLipid raftCerebral CortexRegulation of gene expressionTransforming Growth Factor alphaRatsCell biologyCaveolin 2Animals NewbornExcitatory Amino Acid Transporter 2Gene Expression RegulationNeurologyAstrocytesCaveolin 1Signal TransductionGlia
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Functional interaction of estrogen receptor α and caveolin isoforms in neuronal SK-N-MC cells

2003

Estrogen receptors (ERs) are expressed in neuronal cells and exhibit a wide variety of activities in the central nervous system. The actions of ERs are regulated in a hormone-dependent manner as well as by a number of co-activators and -repressors. A recently identified co-activator of ERalpha is caveolin-1 which has been shown to mediate the ligand-independent activation of this steroid receptor. In the present study we have demonstrated that neuronal SK-N-MC cells lacking functional ERalpha show high levels of caveolin-1/-2 specific transcripts and proteins. Ectopic expression of ERalpha in SK-N-MC cells leads to the transcriptional suppression of caveolin-1 and -2 genes. This silencing e…

Endocrinology Diabetes and MetabolismCaveolin 1Clinical BiochemistryEstrogen receptorBiologyLigandsCaveolinsMethylationModels BiologicalBiochemistryHistone DeacetylasesEstrogen-related receptor alphaEndocrinologyTumor Cells CulturedHumansProtein IsoformsPromoter Regions GeneticDNA Modification MethylasesMolecular BiologyEstrogen receptor betaNeuronsEstrogen Receptor alphaBrainCell BiologyChromatinHormonesChromatinReceptors EstrogenCaveolin 1DNA methylationCancer researchMolecular MedicineCpG IslandsEstrogen-related receptor gammaEstrogen receptor alphaProtein BindingThe Journal of Steroid Biochemistry and Molecular Biology
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Internalization of Echovirus 1 in Caveolae

2002

ABSTRACT Echovirus 1 (EV1) is a human pathogen which belongs to the Picornaviridae family of RNA viruses. We have analyzed the early events of infection after EV1 binding to its receptor α2β1 integrin and elucidated the route by which EV1 gains access to the host cell. EV1 binding onto the cell surface and subsequent entry resulted in conformational changes of the viral capsid as demonstrated by sucrose gradient sedimentation analysis. After 15 min to 2 h postinfection (p.i.) EV1 capsid proteins were seen in vesicular structures that were negative for markers of the clathrin-dependent endocytic pathway. In contrast, immunofluorescence confocal microscopy showed that EV1, α2β1 integrin, and …

IntegrinsReceptors CollagenEchovirusmedia_common.quotation_subjectCaveolin 1ImmunologyIntegrinCaveolaemedicine.disease_causeCaveolinsMicrobiologyClathrin03 medical and health sciencesCapsidVirologyCaveolaeCaveolinEnterovirus InfectionsTumor Cells CulturedmedicineAnimalsHumansInternalization030304 developmental biologymedia_common0303 health sciencesMicroscopy Confocalbiology030302 biochemistry & molecular biologyMolecular biologyClathrinEnterovirus B HumanVirus-Cell InteractionsCell biologyMicroscopy ElectronViral replicationInsect ScienceCaveolin 1biology.proteinRabbitsbeta 2-MicroglobulinJournal of Virology
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